所有产品分类
所有产品
信号通路
- Angiogenesis/Protein Tyrosine Kinase
- Apoptosis Pathway
- Cell Cycle/DNA Damage
- Epigenetics
-
GPCR/G Protein
- Urotensin Receptor
- Melatonin Receptor
- Neurotensin Receptor
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- Oxytocin Receptor
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- Imidazoline Receptor
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- Smoothened/Smo
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- Microbiology/Virology
-
Neuro Signaling Pathway
- Imidazoline Receptor
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- nAChR
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-
Protease/Metabolic Enzyme
- COMT
- 11β HSD
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- Pyruvate Kinase
- Mitochondrial Metabolism
- ALDH
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- Phosphatase
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- RAAS/ACE
- Caspase
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- HCV Protease
- HSP
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- Factor Xa
- DPP
- Procollagen C Proteinase
- Integrase
- Phosphodiesterase/PDE
- Phospholipase
- LXR
- Proteasome
- Cytochrome P450
- TGF beta/Smad
- Wnt/Stem Cell
- Others Pathway
研究领域
- Metabolic Disease
- Neurological Disease
- Endocrinology
- Diabetes
- Cardiovascular Disease
- Immunology
- Allergy
- Inflammation
- Infection
- Cancer
天然产物
抗体
多肽
催化剂
杂质对照品
医药中间体
基础原料
c Myc
结构式 | 货号 | 产品名称 | CAS号 |
---|
BCP41938 | NY2267 新 | 886053-73-2 | |
NY2267 is a disruptor of Myc-Max interaction, with an IC50 of 36.5 μM.
|
BCP40015 | EN4 新 | 1197824-15-9 | |
EN4 is a functional Covalent Ligand Targeting an Intrinsically Disordered Cysteine within MYC.
|
BCP34062 | MYCi975 新 | 2289691-01-4 | |
MYCi975 is an orally active MYC inhibitor, which disrupts MYC/MAX interaction, promotes MYC T58 phosphorylation and MYC degradation, and impairs MYC driven gene expression.
|
BCP34061 | MYCi361 新 | 2289690-31-7 | |
MYCi361 is a MYC inhibitor with the Kd of 3.2 μM for binding to MYC.
|
BCP08097 | 10058-F4 新 | 403811-55-2 | |
10058-F4 is a specific c-Myc inhibitor that disrupts the association between the c-Myc and Max, inhibits HL60 cell growth with IC50 of 51 uM.
|
BCP29977 | CBL 0137 新 | 1197996-80-7 | |
CBL0137 is a metabolically stable curaxin that activates p53 with an EC50 value of 0.37 μM and inhibits NF-κB with an EC50 of 0.47 μM.
|
BCP09223 | 10074-G5 | 413611-93-5 | |
10074-G5 is an inhibitor of c-Myc-Max dimerization with an IC50 of 146 μM.
|
BCP29132 | ML-327 | 1883510-31-3 | |
ML327 is a blocker of MYC which can also de-repress E-cadherin transcription and reverse Epithelial-to-Mesenchymal Transition (EMT).
|